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Matthieu St-Jean, Jessica MacKenzie-Feder, Isabelle Bourdeau, and André Lacroix

( 1 ); during pregnancy, primary adrenal adenoma, adrenocortical carcinoma, bilateral macronodular adrenal hyperplasia (BMAH) or primary pigmented nodular adrenal disease (PPNAD) represent 50–60% of CS cases ( 1 , 2 ). It was suggested that androgen

Open access

Ya-Wun Guo, Chii-Min Hwu, Justin Ging-Shing Won, Chia-Huei Chu, and Liang-Yu Lin

) . The two most common aetiologies of ACTH-independent Cushing’s syndrome are adrenal adenoma and carcinoma. Adrenal adenomas are responsible for approximately 10–15% of cases of Cushing’s syndrome, and adrenal carcinomas are responsible for <5% of cases

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Vasileios Chortis, Christine J H May, Kassiani Skordilis, John Ayuk, Wiebke Arlt, and Rachel K Crowley

use of cross-sectional imaging ( 1 ). Although the majority of AIs represent benign adenomas, a substantial minority consist of malignant tumours, including adrenocortical carcinomas, phaeochromocytomas and adrenal metastases from other primary

Open access

Vivienne Yoon, Aliya Heyliger, Takashi Maekawa, Hironobu Sasano, Kelley Carrick, Stacey Woodruff, Jennifer Rabaglia, Richard J Auchus, and Hans K Ghayee

. International Journal of Surgical Pathology . 20 : 316 – 319 .( doi:10.1177/1066896911427036 ) 22071263 Solis-Lopez DR Rodriguez-Hernandez Z & Solis-Lopez DH 2010 Incidental adreno-cortical adenoma, why surgery? a case

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Rémi Goupil, Martin Wolley, Jacobus Ungerer, Brett McWhinney, Kuniaki Mukai, Mitsuhide Naruse, Richard D Gordon, and Michael Stowasser

perioperative hydrocortisone supplementation followed by several weeks of cortisone acetate (with doses gradually reducing to zero). Macroscopically, the adrenal gland was composed of a large adenoma within an enlarged multinodular gland ( Fig. 1 A). Pathology

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Tohru Eguchi and Shozo Miyauchi

) demonstrated that immunohistochemistry of the human normal adrenal cortex for Aldosterone synthase (CYP11B2) may provide a pathological confirmatory diagnosis of adrenocortical adenomas, but we cannot investigate the immunohistochemistry of CYP11B2. In bone

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Yasufumi Seki, Satoshi Morimoto, Naohiro Yoshida, Kanako Bokuda, Nobukazu Sasaki, Midori Yatabe, Junichi Yatabe, Daisuke Watanabe, Satoru Morita, Keisuke Hata, Tomoko Yamamoto, Yoji Nagashima, and Atsuhiro Ichihara

adrenal expression of some hormone receptors. Aberrant adrenal expression of luteinizing hormone (LH) receptor has been reported in aldosterone-producing adenoma (APA) ( 2 ). It has also been reported that LH increased aldosterone secretion of APA in an

Open access

Skand Shekhar, Rasha Haykal, Crystal Kamilaris, Constantine A Stratakis, and Fady Hannah-Shmouni

cases reported in the literature ( 2 ). We report a case of PA in pregnancy due to an aldosterone producing adrenocortical adenoma that was successfully treated with unilateral adrenalectomy in the second trimester after the failure of medical therapy

Open access

Sophie Comte-Perret, Anne Zanchi, and Fulgencio Gomez

during the investigation of hypertension (1) (2) , as in the patient described here. Hypertension in our patient was largely due to excess adrenocortical secretion, as corroborated by the response to medical inhibition of adrenal steroidogenesis

Open access

Pia T Dinesen, Jakob Dal, Plamena Gabrovska, Mette Gaustadnes, Claus H Gravholt, Karen Stals, Judit Denes, Sylvia L Asa, Márta Korbonits, and Jens O L Jørgensen

may be difficult to visualize; in contrast, macroadenomas (maximal diameter >10 mm) comprise only 4–10% of diagnosed patients (1) . The etiology and pathogenesis of corticotrope adenomas remain unclear and the majority appears to be sporadic. In a