Search Results

You are looking at 1 - 10 of 21 items for :

  • Diabetes insipidus - neurogenic/central x
Clear All
Open access

Alicia R Jones, Alan McNeil, Christopher Yates, Bala Krishnamurthy and Peter S Hamblin

Background The majority of adult patients with central diabetes insipidus (DI) have abnormalities on pituitary magnetic resonance imaging (MRI), with loss of the posterior pituitary high-intensity signal and pituitary stalk thickening being

Open access

Danielle R Bullock, Bradley S Miller, H Brent Clark and Patricia M Hobday

hypopituitarism and/or central diabetes insipidus and review of the literature . European Journal of Endocrinology 2013 170 161 – 172 . ( https://doi.org/10.1530/EJE-13-0642 ) 8 Sosa GA Bell S Christiansen SB Pietrani M Glerean M Loto M Lovazzano S

Open access

Raluca Maria Furnica, Julie Lelotte, Thierry Duprez, Dominique Maiter and Orsalia Alexopoulou

water deprivation test confirmed the diagnosis of central diabetes insipidus. The hormonal profile was otherwise normal ( Table 1 ). A new pituitary MRI showed a stable symmetric but heterogeneous enlargement of the pituitary gland and a loss of the

Open access

Taieb Ach, Perrine Wojewoda, Flora Toullet, Roxane Ducloux and Véronique Avérous

-small-cell lung cancer . Clinical Lung Cancer 2007 8 565 – 567 . ( https://doi.org/10.3816/CLC.2007.n.044 ) 17 Granata A Figura M Gulisano S Romeo G Sicurezza E Failla A Scuderi R . Central diabetes insipidus as a first manifestation of

Open access

Despoina Manousaki, Cheri Deal, Jean Jacques De Bruycker, Philippe Ovetchkine, Claude Mercier and Nathalie Alos

; immediately post-surgery (C) and 6 months post-surgery (D). We note the absence of the bright spot of the posterior pituitary on all four MRI scans. This can be predictive of a permanent central diabetes insipidus although specificity decreases with age

Open access

Ramesh Srinivasan, Stephen Ball, Martin Ward-Platt, David Bourn, Ciaron McAnulty and Tim Cheetham

Background Familial cranial diabetes insipidus (FCDI) is a rare inherited disorder that may have an autosomal dominant inheritance pattern. Polyuria and polydipsia typically develop in childhood due to a progressive decline in AVP secretion

Open access

Snezana Burmazovic, Christoph Henzen, Lukas Brander and Luca Cioccari

inadequate insulin therapy. Moreover, HHS is commonly seen in older patients with type 2 diabetes who have compromised water intake due to underlying medical conditions. Central diabetes insipidus (CDI) is characterized by decreased secretion of

Open access

Florence Gunawan, Elizabeth George and Adam Roberts

, nausea, weight loss and significant polyuria and nocturia. Central diabetes insipidus was diagnosed on the basis of symptoms in the context of pituitary insult and sodium of 149 mmol/L (135–145 mmol/L). No formal 24-h urine volume quantification, serum or

Open access

Lourdes Balcázar-Hernández, Guadalupe Vargas-Ortega, Yelitza Valverde-García, Victoria Mendoza-Zubieta and Baldomero González-Virla

edema but a mild ventricular dilatation ( Fig. 1 ). The diagnosis of hypopituitarism and central diabetes insipidus was established. We initiated the approach of suprasellar lesion. Figure 1 Magnetic resonance image of brain with coronal and

Open access

Aishah Ekhzaimy, Afshan Masood, Seham Alzahrani, Waleed Al-Ghamdi, Daad Alotaibi and Muhammad Mujammami

Background Central diabetes insipidus (CDI), a rather disabling and severe disease characterized by polyuria and polydipsia, is caused by defects of the posterior pituitary, resulting in deficiency of the antidiuretic hormone, vasopressin ( 1