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Angela S Lee and Stephen M Twigg

occur with discontinuation of the opioid therapy. Adrenal insufficiency is a rare but important cause of hypercalcaemia (4) . We report a case of hypercalcaemia subsequently shown to be due to opioid-induced secondary adrenal insufficiency, with the

Open access

Ravikumar Ravindran, Justyna Witczak, Suhani Bahl, Lakdasa D K E Premawardhana and Mohamed Adlan

Background Primary hyperparathyroidism, drugs and malignant disease are common causes of hypercalcaemia in secondary care. However, granulomatous disorders, immobility, familial hypocalciuric hypercalcaemia (FHH) and vitamin D excess are also

Open access

Kirun Gunganah, Ashley Grossman and Maralyn Druce

.79 mmol/l (reference range, 2.2–2.58), PTH level of 4.2 pmol/l (0.6–6.0), low 24 h urine calcium of 0.3 mmol/l and a urine calcium:creatinine ratio of <0.03. A clinical and biochemical diagnosis of familial hypocalciuric hypercalcaemia (FHH) was made

Open access

Jane J Tellam, Ghusoon Abdulrasool and Louise C H Ciin

hypercalcaemia by his GP. He was symptomatic with a 1-month history of fatigue and light-headedness. He denied nausea, polyuria, polydipsia, myalgias, arthralgias, abdominal pain, constipation, flank pain and his mentation was intact. Dietary calcium and vitamin

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Nirusha Arnold, Victor O’Toole, Tien Huynh, Howard C Smith, Catherine Luxford, Roderick Clifton-Bligh and Creswell J Eastman

Background Hypercalcaemia presenting initially during pregnancy or the postpartum period is a rare problem creating major challenges in diagnosis and management and may be responsible for a high rate of maternal and foetal complications ( 1

Open access

Mawson Wang, Catherine Cho, Callum Gray, Thora Y Chai, Ruhaida Daud and Matthew Luttrell

Background Milk-alkali syndrome (MAS) is characterised by the triad of hypercalcaemia, renal impairment and metabolic alkalosis due to the consumption of calcium and absorbable alkali. First described at the turn of the twentieth century, when

Open access

Mona Abouzaid, Ahmed Al-Sharefi, Satish Artham, Ibrahim Masri, Ajay Kotagiri and Ashwin Joshi

presentation An 82-year-old patient was referred to the endocrinology clinic due to hypercalcaemia. His past medical history included dyslipidaemia, osteoarthritis and cognitive impairment. He lived alone, and his medications included atorvastatin

Open access

K Nadarasa, M Bailey, H Chahal, O Raja, R Bhat, C Gayle, A B Grossman and M R Druce

management of parathyroid carcinoma focuses on control of hypercalcaemia, commonly achieved with the use of volume expansion, occasional loop diuretics and bisphosphonate therapy (1) (2) . Unfortunately, the efficacy of these treatments is limited as

Open access

Aisling McCarthy, Sophie Howarth, Serena Khoo, Julia Hale, Sue Oddy, David Halsall, Brian Fish, Sashi Mariathasan, Katrina Andrews, Samson O Oyibo, Manjula Samyraju, Katarzyna Gajewska-Knapik, Soo-Mi Park, Diana Wood, Carla Moran and Ruth T Casey

Background Primary hyperparathyroidism is characterised by hypercalcaemia in the context of a raised or inappropriately normal PTH. It is important to note that the physiological changes of pregnancy can mask its diagnosis, as hypoalbuminaemia

Open access

Lorena Arnez and Victor Lawrence

delivery depending on stage and growth ( 4 ). Case presentation A 40-year-old woman in the 25th week of her third pregnancy was hospitalised and referred to endocrinology services with symptomatic hypercalcaemia after presenting with polydipsia