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Open access

Katsumi Taki, Takahiko Kogai, Junko Sakumoto, Takashi Namatame, and Akira Hishinuma

reabsorption. Germ-line inactivating mutations of CASR cause persistent hypercalcemia with inappropriately normal or elevated PTH along with low urinary calcium extraction, which results in three phenotypes that are usually dependent on an apparent gene

Open access

Nikolaos Asonitis, Eva Kassi, Michalis Kokkinos, Ilias Giovanopoulos, Foteini Petychaki, and Helen Gogas

Background Hypercalcemia of malignancy affects up to 20% of cancer patients during the course of their disease ( 1 ). The majority of cases are humoral in etiology and related to parathyroid hormone-related protein (PTHrP) ( 2 ).The treatment

Open access

Anand Gandhi, Mike Mortensen, Sonie Sunny, Pawarid Techathaveewat, Jerome Targovnik, and Mahmoud Alsayed

Background This case highlights the diagnosis and management of an uncommon cause of hypercalcemia that may often be overlooked by other more common etiologies. It may best be considered after more prevalent conditions that induce

Open access

Arshpreet Kaur and Stephen J Winters

-significant changes in calcium and sodium levels (4) . We present a patient with type 2 diabetes (T2DM) treated with canagliflozin who developed severe hypercalcemia and subsequent hypernatremia following excessive calcium ingestion and diabetic ketoacidosis

Open access

Katsuo Tao, Midori Awazu, Misa Honda, Hironori Shibata, Takayasu Mori, Shinichi Uchida, Tomonobu Hasegawa, and Tomohiro Ishii

dehydration. NDI can be congenital or acquired. Congenital NDI is inherited as an X-linked trait due to AVP receptor-2 ( AVPR2 ) gene abnormality or autosomally due to aquaporin-2 ( AQP2 ) gene abnormality. Acquired NDI is caused by hypercalcemia, hypokalemia

Open access

Reiner Jumpertz von Schwartzenberg, Ulf Elbelt, Manfred Ventz, Knut Mai, Tina Kienitz, Lukas Maurer, Thomas Rose, Jens C Rückert, Christian J Strasburger, and Joachim Spranger

) . Thus, large tumors and severe hypercalcemia indicate higher risk for parathyroid cancer. If parathyroid cancer exists, en bloc resection of the tumor with centrocervical lymphadenectomy is the recommended treatment. Nonetheless, cancer is not suspected

Open access

Carine Ghassan Richa, Khadija Jamal Saad, Ali Khaled Chaaban, and Mohamad Souheil El Rawas

, nausea, vomiting, anorexia 10.4 a Hypercalcemia (12.8 a ) nephrolithiasis hypotension SVD 38 weeks hypercalcemia (11.6 a ) Not done medical therapy: calcitonin Lee et al . ( 3 ) 1 spontaneous abortion PP Acute abdominal pain 11.5 a

Open access

E Mogas, A Campos-Martorell, M Clemente, L Castaño, A Moreno-Galdó, D Yeste, and A Carrascosa

Background Hypercalcemia is an uncommon clinical problem in pediatric patients. Etiologies vary with age at diagnosis, and include parathyroid adenoma or hyperplasia, mutations affecting calcium-sensing receptor (CaSR), hypervitaminosis D

Open access

Alejandro García-Castaño, Leire Madariaga, Sharona Azriel, Gustavo Pérez de Nanclares, Idoia Martínez de LaPiscina, Rosa Martínez, Inés Urrutia, Aníbal Aguayo, Sonia Gaztambide, and Luis Castaño

Background Familial hypocalciuric hypercalcemia type I (FHH1, OMIM #145980) is an autosomal dominant disorder transmitted with a high degree of penetrance and characterized by moderately elevated serum calcium concentrations, low urinary

Open access

Andrew R Tang, Laura E Hinz, Aneal Khan, and Gregory A Kline

intermittent, mild hypercalcemia. From age 2–11 years, she was taking a daily multivitamin containing elemental calcium 100 mg, phosphate 10 mg and cholecalciferol 600 IU. Further work-up is summarized in Table 1 and revealed a high-normal calcium of 2