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Ved Bhushan Arya, Jennifer Kalitsi, Ann Hickey, Sarah E Flanagan and Ritika R Kapoor

divided doses) on day 15 of life. Outcome and follow-up Within 48 h of commencing diazoxide, marked hyperglycaemia (highest bedside BG concentration 21.6 mmol/L) developed, which persisted despite weaning high-concentration glucose intravenous

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Dominic Cavlan, Shanti Vijayaraghavan, Susan Gelding and William Drake

development of ketotic hyperglycaemia in our patient. Abrupt GH-mediated derangement of glycaemic control was demonstrated as long ago as 1958, when Luft et al . (4) used cadaveric GH to treat three type 1 diabetic patients with hypopituitarism and

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Akihiko Ando, Shoichiro Nagasaka and Shun Ishibashi

for 1 week. At that time, hyperglycaemia above 16.7 mmol/L (300 mg/dL) was found and treated by intravenous insulin therapy for 1 month. Subsequently, her growth and development were normal without medication. At 13 years of age, she became aware of

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Benjamin G Challis, Chung Thong Lim, Alison Cluroe, Ewen Cameron and Stephen O’Rahilly

MWS commonly present with diarrhoea, dehydration and symptoms associated with severe electrolyte depletion, particularly those related to hypokalaemia. Symptomatic hyperglycaemia and new-onset diabetes are very rare manifestations of MWS. Herein we

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Ploutarchos Tzoulis, Richard W Corbett, Swarupini Ponnampalam, Elly Baker, Daniel Heaton, Triada Doulgeraki and Justin Stebbing

hospitalisation, she rapidly developed hyperglycaemia and ketosis with blood glucose of about 20 mmol/L and ketones of 2.0 mmol/L, requiring reinstatement of intravenous insulin infusion for a few hours. In the following 2 days, blood glucose readings ranged

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Snezana Burmazovic, Christoph Henzen, Lukas Brander and Luca Cioccari

the first 24 h after admission, her urine output was 3880 mL. Investigation Initial laboratory analysis revealed severe hypernatraemia (serum sodium concentration 161 mmol/L), moderate hypokalaemia (3.2 mmol/L), hyperglycaemia (BGL: 35.4 mmol

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Shivani Patel, Venessa Chin and Jerry R Greenfield

follow-up Blood glucose levels were well controlled on basal bolus insulin. Her hyperglycaemia-associated symptoms had significantly improved. She required 20 units of glargine insulin (Lantus) and 1 unit of insulin aspart (NovoRapid) for every 10 g of

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Chad Bisambar, Andrew Collier, Fraser Duthie and Carron Meney

Background Non-functional duodenal neuroendocrine tumours can present with hyperglycaemia and cause diabetes. This is in the absence of gluconeogenic hormones such as somatostatin and glucagon. Case presentation A previously well 40

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Jose León Mengíbar, Ismael Capel, Teresa Bonfill, Isabel Mazarico, Laia Casamitjana Espuña, Assumpta Caixàs and Mercedes Rigla

, constipation, muscle weakness and weight gain. Physical examination showed puffy face with under-eye oedema and dry skin. Palpation of the neck showed no goitre or cervical pain. Investigation Laboratory analyses showed hyperglycaemia (417 mg/dL) and

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Osamah A Hakami, Julia Ioana, Shahzad Ahmad, Tommy Kyaw Tun, Seamus Sreenan and John H McDermott

insulin levels were low in the presence of hyperglycaemia confirming the diagnosis of T1DM. Noteworthy, the HbA1c at diagnosis was 67 mmol/mol (8.3%). Table 1 Results of investigation. Investigation Value Reference range