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Ismael Capel, Elisabet Tasa-Vinyals, Albert Cano-Palomares, Irene Bergés-Raso, Lara Albert, Mercedes Rigla, and Assumpta Caixàs

, pulmonary diseases, malignancy, neurologic diseases, chronic kidney disease and thyroid diseases ( 4 ). Moreover, hyperthyroidism – and particularly thyrotoxicosis – is a well-known cause of cardiac complications. Though thyroid-related agents such as

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Ehtasham Ahmad, Kashif Hafeez, Muhammad Fahad Arshad, Jimboy Isuga, and Apostolos Vrettos

scale and have entirely different appearances on radioactive iodine nuclear imaging studies. Although cases of conversion from hyperthyroidism to hypothyroidism are often encountered in clinical practice, it is rare to see conversion of primary

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Wei Lin Tay, Wann Jia Loh, Lianne Ai Ling Lee, and Chiaw Ling Chng

on levothyroxine 75 µg/day (1.6 µg/kg/day). However, she remained biochemically hyperthyroid; thus levothyroxine was tapered over the following eight months to 12.5 µg five times a week, 25 µg two times a week (0.3 µg/kg/day). Table 1 Trend of

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Mauro Boronat

in these cases the diagnosis should be easier, it could be complicated by confusion with primary subclinical hyperthyroidism in occasional patients who simultaneously bear some primary thyroid disorders susceptible to cause autonomous thyroid

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Guadalupe Vargas, Lourdes-Josefina Balcazar-Hernandez, Virgilio Melgar, Roser-Montserrat Magriña-Mercado, Baldomero Gonzalez, Javier Baquera, and Moisés Mercado

years later was found to have a mixed FSH- and TSH-producing adenoma resulting in central hyperthyroidism. Case presentation A 19-year-old woman was referred to the Neuroendocrinology Clinic because of headaches and abnormal thyroid function

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Fernando Gomez-Peralta, Pablo Velasco-Martínez, Cristina Abreu, María Cepeda, and Marta Fernández-Puente

Background Liver dysfunction is a complication that can be observed in hyperthyroidism as a result of the use of antithyroid drugs (ATD). Although the overall incidence of hepatotoxicity induced by any antithyroid medication is <0.5%, when we

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Luísa Correia Martins, Ana Rita Coutinho, Mónica Jerónimo, Joana Serra Caetano, Rita Cardoso, Isabel Dinis, and Alice Mirante

destruction and clinical signs of hypothyroidism; finally, the gland is chronically stimulated by agonist antibodies of thyroid-stimulating hormone (TSH) receptor, produced by local B cells, causing hyperthyroidism. However, there is an interrelationship

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Daramjav Narantsatsral, Takagi Junko, Iwayama Hideyuki, Inukai Daisuke, Takama Hiroyuki, Nomura Yuka, Hirase Syo, Morita Hiroyuki, Otake Kazuo, Ogawa Tetsuya, and Takami Akiyoshi

Background Autoimmune thyroiditis is characterized with the release of thyroid hormone resulting in transient hyperthyroidism, frequently followed by a hypothyroid phase before recovery of normal thyroid function. Approximately 5–20% of

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T Min, S Benjamin, and L Cozma

hyperthyroidism and thyroid storm. However, hyperthyroid status is not taken into account while considering anticoagulation for atrial fibrillation. Studies have demonstrated that hyperthyroidism; especially thyroid storm, is associated with an increased risk of

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R D’Arcy, M McDonnell, K Spence, and C H Courtney

avoidance of weight loss supplements. Figure 2 TSH and free T 4 evolution. Figure 3 Free T 3 evolution. Discussion Many patients with hyperthyroidism be it autoimmune or nodular disease will be found to have an