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Chih-Ting Su and Yi-Chun Lin

of IAA presented in IAS ( 3 , 4 ). Herein is a rare case of IA-related recurrent severe hypoglycemic coma probably associated with exogenous insulin analogs, which had similar clinical presentation as IAS. Case presentation A 48-year-old man

Open access

Ved Bhushan Arya, Jennifer Kalitsi, Ann Hickey, Sarah E Flanagan, and Ritika R Kapoor

diazoxide dose to maintain euglycaemia. At age 2.75 years, the patient required further increase in the dose of diazoxide due to occasional hypoglycaemic episodes implying persistence of HH. At last follow-up (age 3.1 years), the patient has normal

Open access

Marina Yukina, Nurana Nuralieva, Maksim Solovyev, Ekaterina Troshina, and Evgeny Vasilyev

associated with decreased blood glucose levels (hypoglycaemia) and neuroglycopenic symptoms and relieved by the administration of glucose. Severe hypoglycaemia is a life-threatening condition due to high risk of hypoglycaemic coma and a fatal outcome

Open access

Nishant Raizada, S H Rahaman, D Kandasamy, and V P Jyotsna

symptoms disappeared after eating food. He had gained 3.5 kg in the last 3 months. There was no history of diabetes mellitus and he had never used oral hypoglycaemic agents or insulin. He was a known hypertensive for the last 15 years and was taking

Open access

Noman Ahmad, Abdulmonem Mohammed Almutawa, Mohamed Ziyad Abubacker, Hossam Ahmed Elzeftawy, and Osama Abdullah Bawazir

insulin release prevents glycogenolysis, gluconeogenesis, lipolysis and ketogenesis; this leads to neuroglycopenia with an absence of alternative source of energy (ketone bodies) exposing the developing brain to hypoglycaemic injury ( 2 ). Congenital

Open access

Khaled Aljenaee, Osamah Hakami, Colin Davenport, Gemma Farrell, Tommy Kyaw Tun, Agnieszka Pazderska, Niamh Phelan, Marie-Louise Healy, Seamus Sreenan, and John H McDermott

levels the patient reported elevated readings ranging from 8 to 22 mmol/L with no hypoglycaemic events. A random blood glucose level checked in the diabetes clinic was elevated at 18 mmol/L. Despite all of the above, however, his HbA1c was 40 mmol