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Siew Hui Foo and Shahada A H Sobah

Histological diagnosis Treatment and outcome PC 39 Female Panhypopituitarism with low cortisol, FSH/LH/oestradiol, FT 4 /TSH; raised prolactin Painful ophthalmoplegia, headache, weight loss, anorexia and raised LDH Pituitary MRI

Open access

Niki Margari, Jonathan Pollock, and Nemanja Stojanovic

Background We describe the case of a female patient who presented with ophthalmoplegia and subsequent pituitary profile and her image revealed a giant invasive prolactinoma compressing the brainstem. It was initially managed with dopamine

Open access

N Siddique, R Durcan, S Smyth, T Kyaw Tun, S Sreenan, and J H McDermott

-up case 3 Three months later the patient’s glycaemic control remained satisfactory, and his ophthalmoplegia had resolved. Discussion of 3 cases The most common presentation of diabetic neuropathy, DSN, is a diffuse and nerve

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Mariana Barbosa, Sílvia Paredes, Maria João Machado, Rui Almeida, and Olinda Marques

, postural dizziness and left partial ophthalmoplegia – Medical 1996 (13) 74 Leuprolide, 7.5 mg 15 min Headache, nausea/vomiting, left ophthalmoplegia, altered mentation, generalized weakness and visual disturbances Stain FSH +, LH +, GH

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Aimee R Hayes, Anthony J O'Sullivan, and Mark A Davies

ophthalmoplegia or diplopia was reported. Remainder of her neurological examination including fundoscopy was normal. Systemic examination was unremarkable and consistent with an uncomplicated pregnancy of 18 weeks of gestation. Investigation Non

Open access

Stephanie Teasdale, Fahid Hashem, Sarah Olson, Benjamin Ong, and Warrick J Inder

, bilateral ophthalmoplegia on all movements, complete on the right, dyscoria and only finger counting for acuity. The visual fields were not documented. Thus, there was a bilateral optic, oculomotor and trochlear nerve palsy, as a right and likely also left

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Gulay Simsek Bagir, Soner Civi, Ozgur Kardes, Fazilet Kayaselcuk, and Melek Eda Ertorer

, ophthalmoplegia, nausea and/or vomiting. The clinical manifestations of PA are related to underlying pathological mechanism, including increased intrasellar pressure, mass effect, compression of neighboring structures and nerves within the cavernous sinus and

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H Joshi, M Hikmat, A P Devadass, S O Oyibo, and S V Sagi

palsy. No other neurological deficit was noted. Visual field assessment was normal. Because of his vascular risk factors, a brain stem ischaemic event causing internuclear ophthalmoplegia was suspected along with the possibility that this could have been

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Ana M Lopes, Josué Pereira, Isabel Ribeiro, Ana Martins da Silva, Henrique Queiroga, and Cláudia Amaral

parasellar invasion causing visual deficits and cranial palsies, respectively. Even though no single clinical feature can establish the diagnosis, the development of DI or ophthalmoplegia from any pituitary lesion should raise the suspicion of metastatic

Open access

Milad Darrat, Mohammad Binhussein, Alan Beausang, Clare Faul, Michael W O’Reilly, Mohsen Javadpour, and Amar Agha

/headache, visual defects and ophthalmoplegia ( 3 ). The commonest endocrine manifestation is diabetes insipidus (70% of the symptomatic patients), followed by anterior hypopituitarism (15% of the symptomatic patients) ( 3 ). Metastases to a pre-existing pituitary